Download PDF by Roger S. Kirby MD FRCS(Urol) FEBU: An illustrated pocketbook of prostatic diseases

By Roger S. Kirby MD FRCS(Urol) FEBU

ISBN-10: 0203324749

ISBN-13: 9780203324745

ISBN-10: 1842142712

ISBN-13: 9781842142714

It is a condensed model of the author's winning Atlas of Prostatic Disease. The emphasis is on a succinct description of prostatic disorder in a mostly illustrative structure. The publication covers the reasons, analysis and staging of prostate melanoma, benign prostatic hyperplasia and prostatitis. The sections on remedy were superior via the addition of algorithms to summarize the decision-making method.

Men's future health is now a big public well-being factor and prostatic affliction may have a tremendous influence at the caliber of lifetime of many males. This e-book will offer a handy precis for urologists, fundamental care physicians and nurse practitioners

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Get An illustrated pocketbook of prostatic diseases PDF

This can be a condensed model of the author's winning Atlas of Prostatic ailment. The emphasis is on a succinct description of prostatic sickness in a mostly illustrative structure. The e-book covers the factors, analysis and staging of prostate melanoma, benign prostatic hyperplasia and prostatitis. The sections on therapy were greater via the addition of algorithms to summarize the decision-making procedure.

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As dedifferentiation occurs due to sequential mutations, however, clonal selection results in an increase in the rate of cell division and the development of local invasion. The TNM (tumors/nodes/metastases) staging system classifies prostate cancers locally as T1–4 (Figure 51). Impalpable tumors, which are now being detected with increasing frequency, are classified as T1A and T1B (according to grade and volume) when identified by transurethral resection (TUR), or as T1C if impalpable, and detected purely on the basis of an elevated PSA and subsequent transrectal ultrasound (TRUS)-guided biopsy.

Critical to this process is the ability of the developing metastases to induce their own blood supply—a process termed angiogenesis (Figure 22). This process depends on a number of proteins known collectively as angiogenesis factors, which appear to be important in conferring metastatic potential21,22. Prostate cancer has a particular proclivity for metastasis to bone. Many tumor-associated factors can stimulate osteoblastic metastases (Figure 23). These include insulin-like growth factors (IGF-1 and IGF-2), transforming growth factors (TGF-β), fibroblast growth factors (FGF-1 and FGF-2), bone morphogenes (BMPs), platelet-derived growth factors (PdGF) and endothelin-1 (ET-1).

34) Significantly, men with larger prostates and (since PSA provides a reflection of total prostate epithelial cell volume) higher PSA values are more likely to develop complications of benign prostatic hyperplasia such as acute urinary retention or require benign prostatic hyperplasia-related surgery. Other risk factors for acute urinary retention include severe symptoms and markedly reduced maximum urinary flow rates34 (Figure 50). The anatomical distribution of the adenoma is not always uniform.

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An illustrated pocketbook of prostatic diseases by Roger S. Kirby MD FRCS(Urol) FEBU


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